GENETIC INFLUENCES ON THE ARTERIAL WALL

GENETIC INFLUENCES ON THE ARTERIAL WALL

Source :

• Clinical and Experimental Pharmacology and Physiology [ 0305-1870 ] ; 2007-07.

English descriptors

• KwdEn :
  • Additive effects, Animal models, Aortic, Aortic dilatation, Aortic stiffness, Arterial, Arterial biomechanical properties, Arterial compliance, Arterial distensibility, Arterial phenotype, Arterial rupture, Arterial stiffening, Arterial stiffness, Arterial wall, Arterial wall hypertrophy, Arterial wall material, Arterioscler, Authors journal compilation, Biol, Blackwell publishing asia, Blood pressure, Boutouyrie, Brown norway, Cardiovascular, Cardiovascular events, Cardiovascular mortality, Cardiovascular risk, Carotid, Carotid artery distensibility, Carotid artery stiffness, Carotid stiffness, Central pulse pressure, Circumferential wall stress, Collagen, Coronary artery disease, Coronary heart disease, Determinant, Elastic lamellae, Elastin, Elastin network, Essential hypertension, Essential hypertensive patients, Extracellular matrix, Gene expression, Gene polymorphisms, Genetic, Genetic component, Genetic factors, Growth factors, Heart rate, Heritability, Hypertens, Hypertension, Hypertensive, Hypertensive patients, Hypertensive rats, Hypertensive subjects, Independent predictor, Kingwell, Laurent, Local pulse pressure, Major cause, Marfan, Marfan syndrome, Matrix, Matrix genotype, Matrix proteins, Mechanical adaptation, Mechanical stress, Monogenic disease, Monogenic diseases, Mouse model, Muscle cells, Pierre boutouyrie, Polymorphism, Precise characterization, Predictive value, Probe sets, Promoter polymorphism, Protein expression, Pulse pressure, Recent study, Residual phenotypic variance, Risk factors, Rotterdam study, Similar results, Smooth muscle, Smooth muscle cell function, Smooth muscle cells, Stiffness, Syndrome, Thromb, Vascular, Vascular wall, Wall components, Wall thickness, Williams syndrome.
• Teeft :
  • Additive effects, Animal models, Aortic, Aortic dilatation, Aortic stiffness, Arterial, Arterial biomechanical properties, Arterial compliance, Arterial distensibility, Arterial phenotype, Arterial rupture, Arterial stiffening, Arterial stiffness, Arterial wall, Arterial wall hypertrophy, Arterial wall material, Arterioscler, Authors journal compilation, Biol, Blackwell publishing asia, Blood pressure, Boutouyrie, Brown norway, Cardiovascular, Cardiovascular events, Cardiovascular mortality, Cardiovascular risk, Carotid, Carotid artery distensibility, Carotid artery stiffness, Carotid stiffness, Central pulse pressure, Circumferential wall stress, Collagen, Coronary artery disease, Coronary heart disease, Determinant, Elastic lamellae, Elastin, Elastin network, Essential hypertension, Essential hypertensive patients, Extracellular matrix, Gene expression, Gene polymorphisms, Genetic, Genetic component, Genetic factors, Growth factors, Heart rate, Heritability, Hypertens, Hypertension, Hypertensive, Hypertensive patients, Hypertensive rats, Hypertensive subjects, Independent predictor, Kingwell, Laurent, Local pulse pressure, Major cause, Marfan, Marfan syndrome, Matrix, Matrix genotype, Matrix proteins, Mechanical adaptation, Mechanical stress, Monogenic disease, Monogenic diseases, Mouse model, Muscle cells, Pierre boutouyrie, Polymorphism, Precise characterization, Predictive value, Probe sets, Promoter polymorphism, Protein expression, Pulse pressure, Recent study, Residual phenotypic variance, Risk factors, Rotterdam study, Similar results, Smooth muscle, Smooth muscle cell function, Smooth muscle cells, Stiffness, Syndrome, Thromb, Vascular, Vascular wall, Wall components, Wall thickness, Williams syndrome.

Abstract

• https://api.istex.fr/document/20535F56F90BFA188FC25CA9E4C02BCBCBBA359C/fulltext/pdf

Affiliations:

• Australie, France
• Île-de-France
• Paris

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